Gavin Atwood of the Brain Injury Alliance of Colorado sent this to me. Click the link to see more about the event.
The President is behind the new BRAIN Initiative.
The Brain Research through Advancing Innovative Neurotechnologies (BRAIN) Initiative is part of a new Presidential focus aimed at revolutionizing our understanding of the human brain. By accelerating the development and application of innovative technologies, researchers will be able to produce a revolutionary new dynamic picture of the brain that, for the first time, shows how individual cells and complex neural circuits interact in both time and space. Long desired by researchers seeking new ways to treat, cure, and even prevent brain disorders, this picture will fill major gaps in our current knowledge and provide unprecedented opportunities for exploring exactly how the brain enables the human body to record, process, utilize, store, and retrieve vast quantities of information, all at the speed of thought.
As part of the BRAIN Initiative, Scientists at the University of California, San Francisco (UCSF) and Massachusetts General Hospital are leading the effort to devise brain implants to help veterans overcome post traumatic stress disorder or PTSD.
Roughly 2.8 million men and women have served in the wars in Iraq and Afghanistan, and it’s estimated that up to 20 percent of those individuals will suffer from post-traumatic stress disorder upon returning home.
In light of this sobering statistic, the Defense Advanced Research Projects Agency (DARPA) has embarked on a 5-year, $70 million project to develop electronic devices that can be implanted in brains to treat PTSD and other psychological problems faced by military personnel. The new devices would both monitor and stimulate specific neural circuits in order to train the brain to function correctly.
Research shows that Magnetic Resonance Imaging (MRI) helps pinpoint the part of the brain that makes us dizzy. The brains of people who experience chronic dizziness and other symptoms after concussion show more white matter damage visible on MRI. The findings suggest that information provided by MRI can speed the onset of effective treatments for concussion patients.
The Craig Hospital Research Department is conducting an online study.
The link below will take you to a survey about advocacy. The survey is designed for people who have had a brain injury as well as for family members/close friends who help advocate for someone who has had a brain injury.
The survey is completely anonymous. We will have no way to link your responses to your email address. It will take about 15-20 minutes to complete.
Your participation will allow us to evaluate programs that empower people with the skills of self-advocacy after brain injury.
Thank you for making a difference in the lives of people with brain injury!
You have to live under a rock to not know about how Traumatic Brain Injury is affecting sports and athletes. Football, Boxing, anything involving getting hit in the head by another person or object are all part of the group.
Concussion rates in US high-school athletes more than doubled between 2005 and 2012, according to a new national study using data on nine team sports.
Researchers suspect the upward trend in reported concussions reflects increased awareness. That's right. The concussions were always there. We have just become better at recognizing them.
Overall, the rate increased from .23 to .51 concussions per 1,000 athlete exposures. An athlete exposure is defined as one athlete participating in one competition or practice.
Traumatic brain injury is a risk factor for epilepsy, though the relationship is not understood. A new study in mice, published in Cerebral Cortex, identifies increased levels of a specific neurotransmitter as a contributing factor connecting traumatic brain injury (TBI) to post-traumatic epilepsy. The findings suggest that damage to brain cells called interneurons disrupts neurotransmitter levels and plays a role in the development of epilepsy after a traumatic brain injury.
Since I am from Las Vegas, new gambling addiction research is interesting.
Researchers from Cambridge University believe hyperactivity in the brain's insula could lead to problem gambling; future treatments could focus on reducing this hyperactivity. Gambling is a widespread activity: 73% of people in the UK report some gambling involvement in the past year and around 50% play games other than the National Lottery. For a small proportion of players (around 1-5%), their gambling becomes excessive, resulting in features seen in addiction. Problem gambling is associated with both debt and family difficulties as well as other mental health problems like depression.
During gambling games, people often misperceive their chances of winning due to a number of errors of thinking called cognitive distortions. For example, 'near-misses' seem to encourage further play, even though they are no different from any other loss. In a random sequence like tossing a coin, a run of one event (heads) makes people think the other outcome (tails) is due next; this is known as the 'gambler's fallacy'.
There is increasing evidence that problem gamblers are particularly prone to these erroneous beliefs. In this study, the researchers examined the neurological basis of these beliefs in patients with injuries to different parts of the brain.
Scientists and researchers have uncovered how inflammation and lack of oxygen work together to cause brain damage in conditions such as stroke and Alzheimer's disease.
The discovery, published today in Neuron, brings researchers a step closer to finding potential targets to treat neurodegenerative disorders.
Chronic inflammation and hypoxia, or oxygen deficiency, are hallmarks of several brain diseases, but little was known about how they contribute to symptoms such as memory loss.
Since the brain uses most of the oxygen it is usually damaged when there is hypoxia.
Gavin Atwood has asked me to forward this Brain Injury Event on to my readers. I have participated with the Brain Injury Alliance of Colorado previously and recommend this event.
Even the mildest form of a traumatic brain injury, better known as a concussion, can deal permanent, irreparable damage.
Now, an interdisciplinary team of researchers at the University of Pennsylvania is using mathematical modeling to better understand the mechanisms at play in this kind of injury, with an eye toward protecting the brain from its long-term consequences.
Their recent findings, published in the Biophysical Journal, shed new light on the mechanical properties of a critical brain protein and its role in the elasticity of axons, the long, tendril-like part of brain cells. This protein, known as tau, helps explain the apparent contradiction this elasticity presents. If axons are so stretchy, why do they break under the strain of a traumatic brain injury?
Tau's own elastic properties reveal why rapid impacts deal permanent damage to structures within axons, when applying the same force more slowly causes them to safely stretch. This understanding can now be used to make computer models of the brain more realistic and potentially can be applied toward tau-related diseases, such as Alzheimer's.
My friend, Gavin Atwood, asked me to share this event with my readers. I have known Gavin for several years and am familiar with his work at the Colorado Brain Injury Alliance.
The Brain Injury Alliance of Oregon has asked me to forward the event below to my readers. I have participated and taught in programs for this group previously and recommend this event.
Being awake at night and dozing during the day can be a distressing early symptom of Alzheimer's disease, but how the disease disrupts our biological clocks to cause these symptoms has remained elusive.
Now, scientists from Cambridge have discovered that in fruit flies with Alzheimer's the biological clock is still ticking but has become uncoupled from the sleep-wake cycle it usually regulates. The findings - published in Disease Models & Mechanisms - could help develop more effective ways to improve sleep patterns in people with the disease.
People with Alzheimer's often have poor biological rhythms, something that is a burden for both patients and their carers. Periods of sleep become shorter and more fragmented, resulting in periods of wakefulness at night and snoozing during the day. They can also become restless and agitated in the late afternoon and early evening, something known as 'sundowning'.
Biological clocks go hand in hand with life, and are found in everything from single celled organisms to fruit flies and humans. They are vital because they allow organisms to synchronise their biology to the day-night changes in their environments.